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Please use this identifier to cite or link to this item: http://tdudspace.texicon.in:8080/jspui/handle/123456789/686
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dc.contributor.authorThomas, Pavana-
dc.contributor.authorKrishna, Sudhir-
dc.contributor.authorSrivastava, Sweta-
dc.contributor.authorPranatharthi, Annapurna-
dc.date.accessioned2025-05-15T07:03:44Z-
dc.date.available2025-05-15T07:03:44Z-
dc.date.issued2019-
dc.identifier.urihttp://tdudspace.texicon.in:8080/jspui/handle/123456789/686-
dc.description.abstractRadioresistance remains a challenge to the successful treatment of various tumors. Intrinsic factors like alterations in signaling pathways regulate response to radiation. RhoC, which has been shown to modulate several tumor phenotypes has been investigated in this report for its role in radioresistance. In vitro and clinical sample-based studies have been performed to understand its contribution to radiation response in cervical cancer and this is the first report to establish the role of RhoC and its effector ROCK2 in cervical cancer radiation response.en_US
dc.language.isoenen_US
dc.publisherJournal of Experimental & Clinical Cancer Researchen_US
dc.subjectRadioresistanceen_US
dc.subjectDNA repairen_US
dc.subjectRhoC ROCK2en_US
dc.subjectCervical canceren_US
dc.titleRhoC regulates radioresistance via crosstalk of ROCK2 with the DNA repair machinery in cervical canceren_US
dc.typeArticleen_US
Appears in Collections:Researcher/Student Publications

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